Postdoctoral Researcher: Aging & Mitochondria–Lysosome Crosstalk

Buck Institute; Novato (CA); On-site; USD 74,000 - 81,000; Full time

Benefits offered by this job

Medical/dental/vision coverage

Paid time off

401(k) employer contribution

Student debt repayment option

Job summary

The Buck Institute for Research on Aging in Novato, CA invites applications for a Postdoctoral Researcher to study mitochondria–lysosome interactions in aging and cellular senescence. The role combines cell biology, advanced imaging, and quantitative analysis within a collaborative, multidisciplinary Zhou Lab team.

We explore organelle communication, high-content imaging, and AI-driven analysis to understand aging-related dysfunction and test strategies to restore signaling in normal aging and

Qualifications

• PhD in cell biology or related field.
• Track record of first-author publications in related field.
• Ability to work independently and collaboratively in a multidisciplinary research setting.
• Strong verbal and written communication skills.

Responsibilities

• Investigate mitochondria–lysosome crosstalk in aging and cellular senescence.
• Employ cell biology, imaging, and quantitative analysis to study organelle communication.
• Apply genetics and mechanistic perturbation approaches to understand aging phenotypes.
• Collaborate with a multidisciplinary team integrating biology, imaging, and computational methods.

Skills

Independent working

Collaborative work

Strong communication

Multidisciplinary teamwork

Education

PhD in cell biology

First-author publications in related field

Job description

The Buck Institute for Research on Aging in Novato, CA invites applications for a Postdoctoral Researcher to study mitochondria–lysosome interactions in aging and cellular senescence. The role combines cell biology, advanced imaging, and quantitative analysis within a collaborative, multidisciplinary Zhou Lab team.

We explore organelle communication, high-content imaging, and AI-driven analysis to understand aging-related dysfunction and test strategies to restore signaling in normal aging and