Research Associate, InflamAb Preclinical Project

Ulster University; Derry/Londonderry; On-site; GBP 30,000 - 37,000; Full time

Benefits offered by this job

Athena SWAN Silver award

Flexible working

Family-friendly environment

Job summary

Ulster University in Derry/Londonderry is inviting applications for a Research Associate to support preclinical development of InflamAb, a novel therapeutic candidate for inflammatory disease. The role involves designing and conducting studies to reveal biological activity, mechanisms, and potential efficacy, with a view to progression to further preclinical work.

The post-holder will generate robust evidence on how InflamAb modulates inflammatory pathways and contribute to translation and

Qualifications

• A first or second-class honours degree (or equivalent) in Biology, Biomedical Sciences, Pharmacy, Biochemistry, or a related discipline, or a Master's degree in a relevant discipline.
• PhD (awarded or near completion with thesis submitted) in a relevant area such as biomedical sciences.
• Previous experience investigating human disease using quantitative research methods.

Responsibilities

• Support preclinical development of InflamAb by designing and delivering experimental studies to investigate its biological activity, mechanism of action and potential therapeutic efficacy.
• Generate robust evidence to establish how InflamAb modulates inflammatory pathways and inform its progression toward further preclinical development.
• Contribute to development and exploitation of innovative approaches to inflammatory disease within the University's research objectives.

Job description

Ulster University in Derry/Londonderry is inviting applications for a Research Associate to support preclinical development of InflamAb, a novel therapeutic candidate for inflammatory disease. The role involves designing and conducting studies to reveal biological activity, mechanisms, and potential efficacy, with a view to progression to further preclinical work.

The post-holder will generate robust evidence on how InflamAb modulates inflammatory pathways and contribute to translation and